Preliminary experiments suggest conolidine could inhibit specific ion channels, decreasing neuronal excitability and restricting pain signals. This mechanism is particularly related in neuropathic suffering, in which abnormal signaling causes persistent discomfort. Additionally, conolidine appears to impact G protein-coupled receptor (GPCR) pathways integral to pain perception. Comput... https://mahatmaw790rix3.bloggactif.com/profile