This work also highlights the cooperation among cell-intrinsic telomere dysfunction–driven molecular pathways and the microbiome in driving the inflammatory reaction. , 2012). The amplified cell proliferation and resistance to cell Demise on ectopic TERT and TERC expression are attenuated by way of repression of p65. Functionally, G1 TERC-null mice were https://friedrichi047icv1.wikiap.com/user