Knocking out hepatic cytochrome P450 triggered a big increase in triptolide concentrations, which aggravated its hepatotoxic results. As a result, circulating AhR levels and microRNA degrees can be used as early warning biomarkers for triptolide-induced cardiotoxicity. Histopathologic analysis confirmed that triptolide-handled mice had significantly less serious kidney disorder, with ... https://triptolide-anticancer-tre02468.blogacep.com/36588723/getting-my-triptolide-to-work